If you reside in an apartment or a house or just want to keep your home free from EMFs There are a variety of ways you can reduce exposure. One of the easiest is to reduce your use of electronic devices. You could also consider EMF block paint to prevent EMF radiation from entering your home. Another easy way to protect your home from EMF radiation would be to use a RF shielding canopy. This is a cloth made of net which contains EMF shielding. emf blockers is utilized to stop EMFs from entering rooms. Another option is to have your home equipped with an electrical enclosure. These devices are called Faraday cages.
A number of studies have proven that the non-ionizing RF EMF has antiproliferative effects on HCC cells. The mechanism of AM RF EMF's anticancer activity in vitro is believed involve down-regulation of cancer stem cells. This could explain the long-term response seen in certain patients suffering from advanced HCC. But, emf blockers for AM EMF's effects on cancer patients is not evident.
The effects of AM RF EMFs on HCC tumour growth in vivo were studied in mice. The tumors were split in three different groups. One group did not have exposure to RF EMF. Second group members were exposed to RF EMF at a frequency that is similar to that used in humans. emf blocking was exposed to RF EMF at HCC-specific modulation frequencies. The effects of HCCMF on tumours was assessed against the effect of RCF. The results showed that the cancers treated with HCCMF had significant shrinkage. However, tumours treated with RCF didn't show evidence of shrinkage in the tumour.

The reason for cancer-specific AM RF EMF may be due to the fact that tumour cells require Cav3*2 T-type voltage calcium channels to promote proliferation and down-regulation. AM RF EMF's antiproliferative effect in HCC cells is controlled by CACNA1H the protein which is responsible for the influx of Ca2+ specific to tumours. The findings suggest that CACNA1H could have more broader implications for the treatment and diagnosis of different cancers.
The tumours of the control group were not exposed to EMF from RF, and fed a standard mouse diet. The tumours in HCCMF HCCMF group were infected with Huh7 cells at the time they were between five and seven weeks old. The tumors were then killed when they showed excessive burden.
The tumours from the three groups showed different growth curves. The tumours treated with HCCMF saw a significant reduction in size of the tumor after eight weeks. However, tumors that were treated using RCF didn't show shrinkage. The difference was highly significant. The tumors treated by RCF had necrosis, which is common when tumors are that are exposed to RCF. There is a possibility that this necrosis is caused by a lack of oxygen in the more invasive tumors.
In sum, the results show that AM RF EMF is a powerful source of anticancer properties in vitro as well as in live. A number of studies have proven it is true that AM RF EMF produces measurable reduction in tumours for HCC patients. The possibility is that the AM EMF triggers these effects because of CACNA1H, a protein involved in the process of tissue-specific Ca2+ influx. Additionally, AM RF EMF may cause a lasting influence on the development of HCC tumours in the vivo.