No matter if you live in a house or apartment or simply want to keep your home free of electromagnetic fields There are plenty of ways you can reduce exposure. One of the most effective is to restrict your electronic device use. emf blockers can also turn to EMF block paint to stop EMF radiation from reaching your house. Another way to shield your home from EMF radiation would be to install a RF shielding canopy. This is a fabric made of net which contains EMF shielding. It's used to stop EMFs from entering rooms. Another alternative is to have your home equipped with an enclosure that is conductive. http://note.pearltrees.com/13949787/5f74121d91af5f39cd84dd85ed5f004e are called Faraday cages.
Numerous studies have demonstrated that the non-ionizing RF EMF has antiproliferative effects in HCC cells. The mechanism behind AM RF EMF's anticancer activity in vitro is believed to involve down-regulation the cancer stem cell. This could be the reason for the long-term responses seen in some patients with advanced HCC. But, the reason for AM EMF's impact on cancer patients is not evident.
Effects from AM RF EMF on HCC tumor growth in vivo was studied in mice. The tumours were divided into three groups. https://te.legra.ph/Info-about-Emf-Blocking-Radiation-04-12-30 did not have exposure to RF EMF. Second group members were exposed RF EMF at the same frequency to the one used by humans. In the third, they were subjected to RF EMF with HCC-specific modulation frequencies. The impact of HCCMF on tumors was evaluated against that of RCF. The results indicated that tumours treated with HCCMF showed significant shrinkage. However, tumours treated with RCF did not show any evidence of shrinkage in the tumour.
The reason for tumour-specific AM RF EMF might be based on the fact that tumour cells require Cav3*2 T-type voltage calcium channels to promote proliferation and down-regulation. AM RF EMF's ability to inhibit proliferation in HCC cells is mediated by CACNA1H the protein that regulates the Ca2+ influx specific to tumors. The results suggest that CACNA1H could have more broader implications for treatment and diagnosis of many cancers.
The tumors in the control group were not exposed to EMF from RF, and fed a normal diet of mice. The tumors of HCCMF HCCMF group were treated with Huh7 cells at the time they were 5 to 7 weeks old. The tumors were then killed in cases of excessive burden.
The tumours in the three groups also showed distinct growth curves. The tumors treated with HCCMF had a significant reduction in the size of the tumour after 8 weeks. However, tumors which were treated by RCF didn't show reduction in size. The difference was significant. The tumors treated by RCF had necrosis, which is typical in tumors that have been that are exposed to RCF. It is possible that this necrosis is caused by a lack of oxygen in the larger tumours.
In conclusion, the findings suggest an AM-RF EMF is a powerful source of anticancer properties in vitro as well as in the vivo. Numerous studies have demonstrated that AM RF EMF produces measurable reduction in tumours in HCC patients. There is a possibility that AM RF EMF triggers these effects because of CACNA1H, a protein that is involved in tissue-specific Ca2+ influx. Additionally, AM RF EMF may have a long-lasting influence on the growth of HCC tumors in living tissue.